1. Sarcoidosis may go away without any treatment.

2. Sarcoidosis with intestinal involvement is the main differential diagnosis in our patient.

3. Sarcoidosis also can produce serious ill effects in the heart, eyes and nervous system.

4. Diagnosis: Sarcoidosis, mild, with perilymphatic nodules.

5. Subpleural nodules are also typical of sarcoidosis.

6. In sarcoidosis there are noncaseating granulomas.

7. Sarcoidosis of nasal ala and lower lip.

8. This appearance is typical of sarcoidosis.

9. Hepatic granulomas most commonly due to sarcoidosis and tuberculosis.

10. Perilymphatic distribution is noted in pneumoconiosis, sarcoidosis, amyloidosis.

11. Conclusion Clinical manifestations of patients with sarcoidosis of the nervous system are varied.

12. In sarcoidosis, nodular interlobular septal thickening reflects the presence of interstitial granulomas.

13. It may also be seen in tuberculosis, sarcoidosis, histoplasmosis, coccidioidomycosis, blastomycosis, ulcerative colitis, and with use of some medications.

14. Objective To analyze chest sarcoidosis on X - ray film.

15. Infiltrative myocardial diseases, including amyloidosis, hemochromatosis, and sarcoidosis, may be associated with atrioventricular block.

16. In patients with sarcoidosis, septal fibrosis can predominate if septal granulomas were active phase of disease.

17. Typically in sarcoidosis is an upper lobe and perihilar predominance and in this case we see the majority of nodules located along the bronchovascular bundle.

18. Met hod: 64 cases of lung sarcoidosis confirmed by pathology were reviwed and anali zed.

19. It is possible that sarcoidosis might be a disease of autoimmune abnormality and antibody of BB in the serum an auto-antibody resulting from the stimuli of certain unknown antigens.

20. No laboratory test or pathological finding can make an absolutely certain diagnosis of sarcoidosis.

21. Not all studies using molecular techniques have produced evidence for M tuberculosis as the pathogen in sarcoidosis.

22. Q: After a recent chest X-ray, my doctor told me I might have sarcoidosis.

23. A somatostatin scan was performed as part of an ongoing research project because the diagnosis sarcoidosis was considered.

24. Nodular thickening of interlobular septa can be seen in lymphangitic carcinomatosis, sarcoidosis, and silicosis.

25. Results No spirochete was found in the lesions of sarcoidosis.

26. Nodular thickening of the peribronchovascular interstitium is particularly common in sarcoidosis and lymphangitic spread of carcinoma.

27. Results:The bioptic tissues were gained wholly from 34 cases of lung diseases including 17 case ofcarcinoma, 5 cases of tuberculosis, one case of sarcoidosis(), and 11 cases of inflammation.

28. Objective To analyse the clinical manifestation and the laboratory features of the sarcoidosis patients with endobronchial miliary lesion.

29. We review the clinical and imaging findings of central nervous system sarcoidosis.

30. Examples of infectious-inflammatory states predisposing to SSS thrombosis are sinusitis, mastoiditis, trauma, and sarcoidosis.

31. Sarcoidosis is an idiopathic systemic disease characterized histologically by the formation of non - caseating granuloma.

32. There was no clinical, radiological or laboratory evidence of autoimmune liver disease, sarcoidosis, lymphoma or tuberculosis.

33. I believe that sarcoidosis explains the cardiac and pulmonary findings in this patient.

34. Associated conditions also include HIV, sarcoidosis, Sjogren syndrome, tuberculosis, mumps, and cat scratch disease. It is rare in pediatric patients.

35. [ Foreman ] We ran some tests and the results point toward sarcoidosis.

36. Conclusion Use of X-ray film is important for the diagnosis of chest sarcoidosis, however, chest sarcoidosis is often neglected in our clinical work.

37. Additional history indicated that the heart transplantation was for sarcoidosis involving the myocardium.