1. Glomerular filtration rate did not vary.

2. Patients with impaired glomerular filtration rate are particularly susceptible to hypermagnesemia when given a magnesium load.

3. The efferent glomerular capillaries form a capillary plexus surrounding the proximal tubules.

4. Changes in glomerular filtration rate and filtered load of sodium are regularly and continuously paralleled by comparable alterations in tubular fluid reabsorption.

5. The fractional excretion of magnesium rises as glomerular filtration rate falls.

6. Glomerular changes are both frequent and severe.

7. The glomerular filtration rate remains relatively constant.

7. try its best to collect and make good sentences.

8. The branches reach to glomerular basal membrane.

9. DNA fragmentation localized to glomerular cells.

10. Furthermore, glomerular damaged showed a significant correlation with tubular DNA damage in proliferatie GN.

11. An increased glomerular staining with Peanut agglutinin lectin, indicate of removal of neuraminic acid, was noted.

12. It is mainly inhibited as glomerular sclerosis, renal interstitial fibrosis and intrarenal vascular sclerosis.

13. Methods Changes relating to glomerular function, structure and glomerular basement membrane ultrastructure were observed.

14. NO was related to both the glomerular hyperfiltration and changes of renal morphology in the early diabetes.

15. Objective To evaluate the value differentiating glomerular hematuria from non-glomerular hematuria with multiple urine acanthocyte(G1 cell)counts.

16. CICs were determined in 92 children with glomerular diseases by PEG precipitation - complement consumption ( PEG - CC ) test.

17. An increased glomerular staining with Peanut agglutinin lectin , indicative of removal of neuraminic acid, was noted.

18. Angiotensin I converting enzyme inhibitors may act in reducing glomerular basement membrane pore size.

19. Data on experimental animals show that it may be the long term consequence of glomerular haemodynamic abnormalities induced by long term hyperglycaemia.

20. This caution can not be overstated in light of the decreased glomerular filtration often present in chronic liver failure.

21. Dynamic renal imaging studies were performed in 132 patients using 99m Tc DTPA and 131 I OIH. Effective renal plasma flow(ERPF) and glomerular filtration rate(GFR) was obtained.

22. Conclusion: Fas and its ligand could play a an role in the injury of human glomerular cell.

23. Conclusions There is early abnormality of renal function in CHF patients without primary renal disorder and the abnormality was featured by impaired glomerular filtration function.

24. Objective To study the expression of lipoprotein lipase(LPL) in human glomerular mesangial cells and the effect of very low-density lipoprotein(VLDL) on the expression of LPL.

25. Results Positive correlations were found between the degree of FN in glomerular mesangium or basement membranes and glomerular lesion grading or tubulointerstitial lesion grading.

26. Results In normal kidney tissues, ILK was mainly located in the glomerular visceral epithelialcells.

27. The primary end point was a change in the fraction of glomerular volume occupied by mesangium in kidney-biopsy specimens.

28. Results consistently demonstrated that DNA colocalized with autoantibodies in glomerular membrane associated EDS.

29. This is a normal glomerulus by light microscopy . The glomerular capillary loops are thin and delicate.

30. Objective : To investigate the mechanism inducing apptosis of human glomerular mesangial cells ( Mcs ) by anti Fas antibody.

31. The renal pathology examination showed that the degree of glomerular proliferation and glomerulosclerosis is significantly attenuated in the two treatment groups.

32. Conclusion: Serum CYS-C is a high sensitive and specific marker in earlier period diagnosis of children kidney diseases and functional lesion of glomerular filtration.

33. Objective To explore the mobile changes of nitric oxide(NO) and the relationship between nitric oxide and glomerular hyperfiltration in experimental diabetic rats.

34. Results Among 42 cases electrophoresis indicated 27 glomerular proteinuria; 15 mixed proteinuria.

35. Magnified portion of a section showing the glomerular capillaries and Bowman's space.

36. Renal interstitium was hyperemic , the glomerular volume became bigger, and renal tubular epithelial cells showed swelling and glassy degeneration.

37. The histological and pathological examination showed that crescent formation of glomerular capsule and necrosis of renal tubule in high dosage group.

38. Objective To evaluate the changes of glomerular filtration rate (GFR), effective renal plasma flow (ERPF) and microalbuminuria in early diabetic nephropathy (DN) and their correlation.

39. What about this topic and the use of S creatinine and glomerular filtrate rate?

40. Other risk factors are smoking, dyslipidemia, proteinuria, glomerular hyperfiltration and dietary factors.

41. The development of severs hypertention in glomerular disease is an ominous s ign.

42. The result suggests examining the morpho- logic features of erythrocytes in the urine with smear staining method can distinguish between glomerular and nonglomerular hematuria.

43. Conclusion: Irbesartan can up-regulate the renal HGF, strengthen its renotrophic function and retard the development of glomerular sclerosis and tubulointerstitial fibrosis.

44. About 15 % of glomerular filtration occurs through the mesangium, with the remainder through the fenestrated epithelium.

45. The frequencies of mesangial proliferation, glomerular nodule lesions, and glomerular sclerosis were higher in DN patients with amylin deposits.

46. Immune complexes nephritis is a typical nephritis model, its pathological change finally lead to glomerular sclerosis.

47. Most of cases were considered as cryoglobulinemia related glomerulonephritis by the ultrastructural findings of glomerular organized deposits.

48. The pathological changes were characterized by formation of antigen-antibody complexes both in the alveoli basement-membrane and the glomerular basement-membrane.

49. Glomerular filtration rate (GFR) and Effective renal plasma flow (ERPF): metered principles and clinical applications.

50. No significant changes were observed in glomerular filtration rate and rennin activity after administration of RDA.

51. A few deteriorated endothelial cells were lifted focally from the RBM and GBM with subendothelial swelling in retinal vessels and glomerular capillaries at 2 weeks and 1 month.

52. Glycosaminoglycans act to restore glycoproteins present in reduced amounts in the glomerular basement membrane and mesangium of diabetic animal models.

53. Objective To study the relationship between the concentration of serum endogenous ouabain (EO) and glomerular diseases.

54. Results According to the images of urine protein electrophoresis, proteinuria can be classified into five types: glomerular, tubular, mixed, over flow, and physiological.

55. The result showed that the occurrence of acanthocyte in the glomerular bleeding(5.5%)was much higher than that in the nonglomerular bleeding(0.6%).

56. Conclusion: Endogenous metanephros origin of glomerular endothelial cells is determined.

57. Concurrent interstitial morphological alterations and hyalinization of afferent and efferent glomerular arterioles also occur.

58. Cast preparation of the glomerular vessels ( glomerulus ) in a renal corpuscle.

59. The changes of polyanion of the glomerular filtration barrier and the morphological changes of epithelial foot processes were studied using an Image Analyzer and stereological techniques.

60. Puromycin aminonucleoside-induced nephrosis is a well-described model of human idiopathic nephrotic syndrome, suppressing expression of integrin expression in cultured glomerular epithelial cells.

61. Glomerular Filtration Rate (GFR) and Effective Renal Plasma Flow (ERPF) were measured by SPECT in 172 patients (192 times) with various renal diseases and 18 normal controls.