(1) Those may be caused by vasospasm and angiostenosis.

(2) Methods: Sixteen cases with symptomatic vasospasm after SAH were treated with isovolemic hemodilution and dobutamine and CDPC.

(3) Preeclampsia-eclampsia is caused by generalized vasospasm, activation of the coagulation system, and changes in autoregulatory systems related to blood pressure control.

(4) INTERVENTION: Vasospasm was confirmed by angiography and transcranial Doppler sonography, and symptoms responded to hypervolemia, hypertension, and hemodilution therapy.

(5) Five of the six patients had angiographically confirmed vasospasm, and one of them later developed a cerebral infarction.

(6) Curtin etal reported ventricular fibrillation in a 42-year-old woman within 3 min of injection, thought to have been induced by vasospasm.

(7) Conclusion The continuous intraarterial infusion papaverine has preventive effects in delayed cerebral vasospasm.

(8) Objective To analyze the effect of cinepazide meleate in treatment of cerebral vasospasm(CVS) using TCD.

(9) Objective To study the preventive effect of azathioprine on chronic cerebral vasospasm ( CVS ) .

(10) CLINICAL PRESENTATION: We present a patient who developed a partial expressive aphasia 9 days after developing meningitis , consistent with cerebral vasospasm, after lumbosacral spinal surgery.

(11) The medicine combination can regulated the contraction and the relaxation of cerebral vasospasm in bidirectional way and has significant curative effect on the angioneurotic headache.

(12) Objective To observe the curative effect of narceine and 3H therapy for the patients with cerebral vasospasm after ruptured intracranial aneurysm.

(13) Calcium antagonists canlunwen 114 prevent vascular smooth muscle contraction caused by the vasospasm.

(14) Objective To evaluate the effect of intrathecal infusion of papaverine for the prevention of vasospasm following subarachnoid hemorrhage ( SAH) .

(15) Conclusions Acupuncting NeiRuan point can regulate pathological ET and NO at early myocardial infarction, and improve diastolic contract function of ischemic micrangium, relieve coronary vasospasm.

(16) Objective To study the cellular apoptosis of symptomatic cerebral vasospasm ( CVS ).

(17) Objective:To investigate the effect of intracisternal nimodipine therapy on cerebral vasospasm in the rabbit double subarachnoid hemorrhage model.

(18) Objective : To observe buflomedil hydrochloride in the prevention and treatment of vasospasm after subarachnoid hemorrhage.

(19) Conclusion Using narceine and 3H therapy is effective for patients with cerebral vasospasm after ruptured intracranial aneurysm.

(20) The subarachnoid hemorrhage ruptured aneurysm is more of an irritant producing vasospasm than a mass lesion.