1. The hypocalcemia is associated with hyperphosphatemia in the absence of bone or renal disease.

2. The other causes of hyperphosphatemia listed in Table 3-11 are rather uncommon.

3. Hematologic studies revealed severe hyperphosphatemia, hypocalcemia and metabolic acidosis.

4. Hyperphosphatemia, a nearly universal complication of kidney failure, is accompanied by hypocalcemia and low serum levels of vitamin D.

5. Phosphate – hyperphosphatemia causes precipitation of serum calcium, leading to hypocalcemia and dysrhythmias.

6. Fatal hyperphosphatemia after oral phosphate overdose in a premature infant.

7. Investigations revealed hypocalcemia, hyperphosphatemia, and a low serum level of intact parathyroid hormone.

8. Tetany, epilepsy, neuro-psychiatrical symptoms, hypocalcemia, hyperphosphatemia were common clinical manifestation. There was also low level of PTH, except pseudohypoparathyroidism.

9. Likewise, the presence of hyperphosphatemia, acidosis, and proteinuria may be misleading depending on the experimental model and diet and may not represent a precise measure of progression.

10. If one follows these guidelines, the risk of hypercalcemia and hyperphosphatemia and their complications is reduced.

11. Administration of calciferol to patients in excess of their daily requirements can cause hypercalcemia, hypercalciuria and hyperphosphatemia.

12. Results:The main abnormal blood chemical features in all cases were hyperkalemia, hyperuricemia, hyperphosphatemia and hypocalcemia and abnormal function of the kidney.

13. Objective To compare the effect and safety on treatment of hyperphosphatemia in hemodialysis patients with calcium acetate and calcium carbonate.

14. Objective: To investigate the clinical characteristics and mechanism of renal failure due to acute hyperphosphatemia following intravenous fructose diphosphate injection.

15. After 7 months'therapy the patient developed progressive weakness, intolerance to hemodialysis,[sentence dictionary] hyperphosphatemia and hypercalcemia.

16. Dietary restriction of phosphate has long been the cornerstone of therapy, but this measure is usually not sufficient to control hyperphosphatemia.