1. An ulcer perforates the duodenal wall.

2. Drugs have been developed to cure some duodenal ulcers.

3. Duodenal biopsy specimens were taken in 50 patients.

4. The main, unsolved clinical problem is chronic duodenal ulcer.

5. In conclusion,() dietary polyunsaturated fatty acids enhance duodenal resistance to acid by potentiation of adaptive cytoprotection.

6. Discussion Duodenal ulcer disease has traditionally been associated with excess secretion of gastric acid.

7. After eradication of H pylori, duodenal ulcers do not usually recur and the associated chronic gastritis gradually disappears.

8. The opportunity should be used to obtain duodenal biopsy specimens.

9. He excluded patients with cystic fibrosis by performing duodenal drainage tests for the measurements of pancreatic enzyme concentrations in the duodenal juice.

10. There was no correlation between duodenal juice protein, enzyme turnover rates, and enzyme secretion.

11. There were eight smokers in the duodenal ulcer group and three in each of the other two groups.

12. A bacterium is identified as the cause for his duodenal ulcer.

13. Eighty unrelated controls, 61 patients with gastric ulcer, and 57 patients with duodenal ulcer were studied.

14. The role of the increased serum gastrin concentration induced by H pylori in the pathogenesis of duodenal ulcer disease is also unknown.

15. Only a minority of patients infected with H pylori will eventually develop a duodenal ulcer.

16. Indeed, there were fewer patients than expected taking NSAIDs, perhaps since intake had been reduced because of chronic duodenal ulcer disease.

17. Treatment failure was defined as evidence by endoscopy of a recurrent duodenal ulcer crater.

18. Thus H pylori infection combined with active ulceration may cause the increased basal acid secretion seen in duodenal ulcer disease.

19. The two patients in whom the lesion was not reached had ulcers located in the inferior and posterior wall of duodenal bulb.

20. Protocol 2 studied the phenomenon of adaptive cytoprotection in response to mild irritation of the duodenal mucosa in the three experimental groups.

21. Furthermore, many studies have shown that the naturally occurring relapse rate of duodenal ulcers is considerably reduced after eradication.

22. This may partly explain the lack of change in the appearance of the duodenal mucosa on serial biopsy in this condition.

23. Many studies have demonstrated that genetic factors contribute to susceptibility to duodenal ulcers.

24. No other model of chronic ulcer shows such morphological and behavioural similarity to the human duodenal ulcer.

25. Another patient had severe inflammation and numerous granulomata on histological examination of duodenal biopsies indicating Crohn's disease of the duodenum.

26. According to previous studies, gastric metaplasia is an almost constant finding in patients with duodenal ulceration.

27. The exaggerated acid response to gastrin can be explained by the increased parietal cell mass present in duodenal ulcer patients.

28. H pylori was not examined because its importance in duodenal ulcer disease was not widely recognised when this study was being planned.

29. In 1949 Lemon and Byrne reported a large series of patients undergoing duodenal aspiration cytology with a test sensitivity of over 70%.

30. They compared the finger and palm prints of 64 healthy adult males and 90 males with duodenal ulcers.

1. A bacterium is identified as the cause for his duodenal ulcer.

31. All carriers of the bacterium, however, do not suffer from duodenal ulcer.

32. Risk factors for delayed duodenal ulcer healing are mentioned in all textbooks as important considerations in the management of duodenal ulcer disease.

33. As a result, the inflamed duodenal mucosa may then become susceptible to acid and this may ultimately lead to ulceration.

34. We conducted such a study in patients with active duodenal ulcer disease.

35. Patients after acute pancreatitis had slower duodenal juice protein and amylase turnover rates but trypsin turnover was not different with controls.

36. A positive provocation test was documented if upper abdominal pain was present with or without nausea after introduction of duodenal contents.

37. In addition, the phospholipid composition of gastric mucosa was compared with that of duodenal mucosa in 10 patients with duodenal ulcer.

38. Gastrin levels may also be elevated by pernicious anemia. duodenal ulcers, and after a meal.

39. Endoscopy in the nine H pylori positive non-uraemic patients showed oesophagitis in one patient and active duodenal ulcer in another.

40. When food leaves the stomach or duodenal during the night, the ulcer is bathed in acid.

41. Decreased concentrations of activated pepsinogen were found in H pylori positive patients only. Duodenal biopsies were tested in 76 patients.

42. Diets rich in polyunsaturated fatty acids enhance the phenomenon of adaptive cytoprotection and render the duodenal mucosa more resistant to acid.

43. Epigastric pain was the main clinical symptom of duodenal ulcer disease: this was experienced by all patients before entering the study.

44. In our previous study, the ratio of gastric to duodenal ulcers was 1.69 in Kinki district where Kyoto Prefecture is located.

45. H pylori positive gastritis, and the combination of active duodenitis and gastric metaplasia were independent predictors of duodenal ulceration.

46. The finding of enhanced fasting gastrin concentrations in H pylori positive subjects and in duodenal ulcer disease can not easily be explained.

47. There are many models of duodenal ulcer - do we need a new one?

48. The absence of a low folate value should not deter the physician from obtaining a duodenal biopsy specimen.

49. Pepsin measurements were not performed in two of the 10 duodenal ulcer patients after treatment.

50. Thus, in addition to duodenal ulcer disease, H pylori eradication may also cure gastric ulcer disease.

51. To determine the extent of gastric metaplasia, multiple biopsy specimens were collected from standardised sites of the duodenal bulb.

52. Both factors may be part of a complex cascade of events that ultimately leads to the development of duodenal ulceration.

53. Laparotomy showed duodenal scarring with enlarged lymph nodes in the pyloric and duodenal areas and white nodules in the liver.

54. Models of mucosal damage in which a noxious agent such as ethanol is employed are simply not relevant to chronic duodenal ulcer.

55. Bonnevie reported that the incidence rate of duodenal ulcer was four times higher than that of gastric ulcer in Copenhagen County.

56. Five out of 10 patients had a positive result on provocation with their duodenal aspirate, developing epigastric pain.

57. Duodenal ulcer patients were asked to stop any antisecretory treatment two weeks before the secretory studies.

58. Phosphatidylglycerol was detectable in patients with chronic atrophic gastritis, but not in controls or in patients with duodenal ulcer.

59. Six patients had gastric ulcers and five had duodenal ulcers.

60. This finding provides support for the belief that adequate treatment of H pylori infection will give longterm protection from duodenal ulcer recurrence.

61. The high pepsinogen C concentrations in patients with duodenal ulcer are probably related to H pylori related chronic active gastritis.

62. Balri etal found that the metabolic clearance rate of G17 was lower in patients with duodenal ulcers than in controls.

63. Discussion Our current results confirm that treatment which both heals duodenal ulcers and eradicates H pylori significantly decreases basal plasma gastrin concentrations.

64. Kothary etal have reported that terminally extended forms of gastrin in conjunction with G14 are more prevalent in duodenal ulcer patients.

65. Each of these abnormalities will increase the exposure of the duodenal mucosa to acid and thus explain its ulceration.

66. The difference between mean rates of secretion of acid and pepsin in control subjects and patients with duodenal ulcer is about 190%.

67. The phosphatidylethanolamine value was higher in duodenal ulcer and lower in chronic atrophic gastritis compared with the control group.

68. Kurata etal reported that duodenal ulcers were diagnosed 2.5 times more frequently than gastric ulcers in Los Angeles, California.

69. After eradication of H pylori in the duodenal ulcer patients both their basal acid output and basal gastrin fell by 50%.

70. The most obvious explanation is that reflux of duodenal content into the stomach after operation is responsible.

71. Hence, we postulated that adaptive cytoprotection maintains a physiological equilibrium between duodenal mucosal resistance and luminal acidity.

72. In 38 patients endoscopy showed focal abnormalities or signs of diffuse inflammation of the duodenal bulb.

73. Five patients had additional selective gastric vagotomy because of excessive gastric acid or a history of duodenal ulcer.

74. Cyclical changes in H pylori infection may cause the variations in basal acid secretion that are seen in duodenal ulcer disease.

75. The aim of our study was to find out if the more potent prokinetic drug cisapride could prevent duodenal ulcer relapse.

76. There was no significant difference in phospholipid composition between antral and duodenal sites in duodenal ulcer patients.

77. Lysolecithin was the smallest component in the duodenal ulcer and chronic atrophic gastritis groups.

78. Peptic ulcer is a convenient term which covers both gastric and duodenal ulcers.

79. If you have many whorls, few loops, patterned palms the chances are you will develop duodenal ulcers.

80. Objective:To study the operative treatment of duodenal diverticulosis.

81. Duodenal atresia:Double bubble and polyhydramnios.

82. Symptomatic duodenal diverticulitis is rare.

83. Conclusion: More attention should be paid to duodenal injury.

84. Drawing ( coronal view ) shows the locations of duodenal fossae.

85. Cysteamine can deplete tissue somatostatin (SRIF) and induce duodenal ulcers in rats.

86. The complications of duodenal ulcer that require surgical management are hemorrhage , perforation, obstruction, and intractability.

87. In summary, vascular aneurismal lesion should be included in the differential diagnosis of a duodenal submucosal tumor-like lesion.

88. The authors reported nursing of duodenal fistula patients treated with balloon catheter isolating digestive juice early operation repair.

89. In a duodenal ulcer, a long post parricidal interval of two to three hours offend exists before pain is apparent and it may then occur immediately before next meal.

90. Objective To evaluate the value of magnetic resonance cholangiopancreatography ( MRCP ) in detecting duodenal diverticulum.

91. Objectives Discussing the effect of highly selective vagotomy (HSV) and repair treatment in perforation of duodenal ulcer.

92. Conclusion:Positive family history, engorgement, smoking, irregular eating, depression and anxiety can induce gastric and duodenal ulcer and thus increase its incidence.

93. Results The observation group 29 cases, 25 cases are independent of success, 4 cases through fiber with nasal endoscopy directly to duodenal or jejunal bowel, normal gastric tube routine.

94. An exploratory operation is very important in the diagnosis of duodenal injury.

95. The strongest association with Helicobacter pylori is with peptic ulceration -- over 85 % of duodenal ulcers.

95. is a sentence dictionary, on which you can find good sentences for a large number of words.

96. Objective:To investigate extent of lesion, grading criteria of nonspecific duodenitis(NSD) and their association with duodenal ulceration.

97. Alpha-chain protein was not demonstrated in the patient's serum, urine, diluted duodenal Juice, and jejunal biopsy tissue.

98. Methods:50 rats received pancreaticoduodenal transplantation using duodenal drainage. 12 dead tats were dissected and the causes for death and the complication were analysed.

99. Method: 25 cases with duodenal diverticulum diagnosis ed and treated near 20 years were reviewed.

100. Objective To assess the association between juxtapapillary duodenal diverticula(JDD) and choledocholithiasis.

101. Objective To probe into the X - ray diagnosis character and clinical significance of duodenal diverticulitis.

102. Objective To evaluate the effect of modi fied highly selective vagotomy for the treatment of perforated duodenal ulcer.

103. Cabbage contains chemicals that help heal both gastric and duodenal ulcers.

104. Conclusion The most frequent cause of the duodenal mucous prominences lesion are chronic duodenitis, then was the polypoid, tumor (16%).

105. Surgical operation confirmed that 46 cases with malrotation of intestines, 24 with congenital duodenal atresia or stenosis (including membranous stenosis) and 15 with annular pancreas.

106. Short and stubby hands and absent thumbs, eye abnormalities including iris dysgenesis, porokeratosis and cataracts, annular pancreas, duodenal stenosis.

107. Conclusion Double bubble sign is most commonly seen in duodenal obstruction.

108. It may merely demonstrate scarring of the duodenal bulb caused by previous ulceration.

109. Objective To investigate the etiology, clinical diagnosis and surgical treatment of the duodenal diverticulum.

110. Objective:To evaluate the role of computed tomography (CT), in diagnosis of the duodenal ampulla cancer.

111. In 41 cases, 7 cases were of stomach and duodenal hemorrhage, 23 cases small intestinal hemorrhage, 11 cases colonorrhagia.

112. Objective To study the early diagnosis of duodenal injury closed abdominal trauma.

113. Conclusions:The repair of ruptured duodenum with simple suture plus duodenostomy and jejunostomy is preferential option for majority of cases of duodenal trauma.

114. Results:To hold the indication strictly, and to choice the method of operation are the most important reasons to treat duodenal diverticulosis successfully.

115. At laparotomy, duodenal dverticulitis and one enterolith obstructing the distal ileum were found.

116. Duodenal diverticulum manifested as hemicycle lesion with gaseous density which had air-fluid level behind pancreatic head with clear border.

117. Objective To construct eukaryotic expression vector of human pancreatic duodenal homeobox 1(PDX-1) gene, and to detect its expression in NIH3T3 cell lines.

118. This is a retrospective summary of the curative effect of highly selective vagotomy for treating 50 Patients with duodenal ulcer.

119. Objective To evaluate the long-term results of extended parietal cell vagotomy (EPCV) for the treatment of acutely perforated duodenal ulcer.

120. An duodenal ulcer is seen in two views on upper endoscopy.

121. Objective To study the efficiency of endoscope-guided Savary bougie dilatation ( ESBD ) for strictured duodenal ulcer.

122. Objective : To investigate the method of diagnosis and treatment of duodenal diverticulum.

123. Objective To evaluate the value of MRI & MRCP in diagnosis of duodenal papillary carcinoma.

124. Conclusions The main etiology of acute pancreatitis in cholecystectomized patients was choledocholithiasis, and the second cause was duodenal papillitis.

125. D: According to your symptoms, it looks like you have duodenal ulcer.

125. is a online sentence dictionary, on which you can find good sentences for a large number of words.

126. Results: In 60 patients, 73 duodenal diverticula were detected, 58 of whom were JPD.

127. Conclusions Combined biliary and duodenal stenting is an effective method palliation of biliary and duodenal obstructions.

128. Gastritis, duodenitis and duodenal ulcer were in the order of incidence in the middle-aged group.

129. Objective To assess the clinical value of duodenal circular drainage operation to treat superior mesenteric artery syndrome(SMAS).

130. Objective To conclude the effect of parietal cell vagotomy for duodenal ulcer and its complications.

131. Pathological examination after endoscopic polypectomy demonstrated mature adipose tissue in the submucosa, consistent with a diagnosis of duodenal lipoma.

132. Multiple diverticulosis of jejunum is often complicated with the duodenal diverticulum, which usually occurs in the proximal and middle section.

133. Objective To summarize the experience of diagnosis and treatment of juxta ampullary duodenal diverticula (JPD).

134. It suggests that BPP may protect duodenal mucosa from ulceration by preventing local SRIF depletion caused by cysteamine .

135. Gastric and duodenal ulcers were noted to evolve from early superficial mucosal disease.

136. The old people with gastric and duodenal ulcer are not suitable to drink tea with limosis in the morning.

137. Individuals with stomach ulcers, duodenal ulcers,() or gallstones should not take devil's claw unless recommended by a health care provider.

138. An effective duodenal drainage and enteral nutrition through jejunostomy are beneficial to healing the injury.

139. Objective: To discuss the value of MSCT in diagnosis of duodenal diverticula .

140. Pregnant women and stomach, duodenal ulcer patients not take it.

141. John BC, John FE . Delayed diagnosis of duodenal rupture. Am J Surg 1996 ; 168:676 - 679.

142. Alimentary tract contrast examination is the first choice for duodenal diseases.

143. Objective To discuss the effects and the main operative techniques of laparoscopic highly selective vagotomy (LHSV) in the treatment of perforating duodenal ulcer.

144. Conclusions In neonatal period, the etiology of intestine obstruction non-operation reason mainly is small intestine atresia, malrotation of intestine, duodenal septum stenosis.

145. Methods: The efficacy of omeprazole and famotidine in treating 82 duodenal ulcer patients were analysed.

146. Objective To evaluate the curative effects of gastroduodenal drainage for serious duodenal injury.

147. Results: All of the 64 cases were clinically cured; there was no duodenal stump rupture, no stomal leak, and no abdominal cavity infection.

148. The rate of duodenal papillitis in non-biliary patients was 37.9%(11/29).

149. Results: There were 6 cases of duodenal atresia, 14 cases of jejunal atresia, 19 cases of ileal atresia and 3 cases of colon atresia in 42 cases of CIA.

150. Conclusion: Multiple diverticulosis of jejunum is often complicated with the duodenal diverticulum, which usually occurs in the proximal and middle section.

151. Objective:To compare and evaluate the short term effect of laparoscopic neoplasty and open neoplasty in the perforated duodenal ulcer.

152. Annular pancreas is a rare congenital abnormality resulting from a fusion anomaly of the bilobed ventral pancreatic bud, with the dorsal bud and associated duodenal migration.

153. Objective To investigate the effect calcitonin gene - related peptide ( CGRP ) 、 endothelin ( ET ) on duodenal ulcer ( DU ).

154. The results showed that duodenal afferent messages were involved in the inhibitory effects of GAO and GMBF response to intragastric peptone during duodenal infusion of hyperosmotic solution.

155. Objective To improve the level of diagnosis and treatment of duodenal injury.

156. Other common gastric diseases were, peptic ulcers (2.53% i. e. , 1.88% for duodenal and 0.49% for gastric ulcers ) and hiatus hernia ( 0.89%).

157. Previous duodenal ulcer but quiescent for years - no contraindications to thrombolysis?

158. Conclusion Juxtapapillary duodenal diverticulum might be one of the causes for choledocholithiasis and recurrent stones.

159. 75 % of postbulbar ulcers occur proximal to the duodenal papilla.

160. Objective: To clarify the relationship between blood group ABO and duodenal ulcer ( DU ) in patients with H. pylori ( Hp ) infection.

161. The main surgical procedure of primary duodenal carcinoma is pancreatoduodenectomy, segmental duodenectomy and simple tumor resection.

162. The duodenal wind sock sign has been described as a typical appearance of an intraluminal duodenal diverticulum (1).

163. Methods Analyze the character of 38 cases duodenal diverticulitis in X-ray, which were confirmed by pathology.

164. Objective To study the long term results of extended parietal cell vagotomy (EPCV) in the treatment of patients with duodenal ulcer and their complications.

165. Superior mesenteric artery syndrome is an uncommon cause of duodenal obstruction.

166. An acute duodenal ulcer is seen in two views on upper endoscopy.

167. Gastritis, duodenal ulcer and duodenitis were in the turn in younger-aged group.