1) The corpus callosum, pyramidal tract, basal ganglia, four stacked body, choroid plexus and brain stem, etc. may shrink as a result of prolonged pressure.

2) Note the contrast enhancement corpus callosum , indicating the probable route of speak of tumor between hemispheres.

3) This operation involved cutting the main fibre tract that joins the two halves of the brain, the corpus callosum.

4) These abilities have been most clearly shown for some epileptics whose corpus callosum has been severed.

5) Objective To investigate whether diffusion indices of the corpus callosum can differentiate relapsing neuromyelitis optica (RNMO) from relapsing-remitting multiple sclerosis (RRMS).

6) The 1HMRS indexes at genu and splenium of corpus callosum, and basal ganglia could serve as effective indexes for the diagnosis of DAI.

7) Results: The parameters analyzed showed no significant difference between the group of patients with focal lesions in the splenium of the corpus callosum and the control group.

8) Methods The imaging findings of 9cases Dysgenesis of the Corpus Callosum diagnosed by computed tomography(CT)and magnetic resonance imaging(MRI)were analysed retrospectively.

9) Figure 1: Axial non contrast CT demonstrates symmetric low attenuation in the temporal-parietal white matter. Note involvement of the splenium of the corpus callosum (red arrows).

10) Head MRI showed infarction of left paraventricular and corpus callosum, and old lacuna infarction of right basal ganglion and pons.

11) MRI demonstrated reduction ,(http:///callosum.html) dysplasia and absence of corpus callosum.

12) Objective To study the CT and MRI findings of Dysgenesis of the Corpus Callosum.

13) Conclusion MRI can directly observe the morphologic construction of the corpus callosum so that MRI should be a first choice in the diagnosis of callosal agenesis.

14) The researchers saw significantly increased activity within the corpus callosum, the area that connects the left and right brain hemispheres, in those with Tourette.

15) Conclusion MRI is the best method to diagnose the dysgenesis of the corpus callosum.

16) The two sides communicate with each other and work together via a complex wodge of neural cabling known as the corpus callosum.

17) Methods: The clinical and CT data of 30 patients with corpus callosum injury were analysed retrospectively.

18) When the anterior horn of lateral ventricles appears, the extended lines of bilateral anterior rami of lateral sulcus intersect on the superior aspect of the genu of corpus callosum by obtuse angle.

19) In normal group and sham-operated group, Microglia in medullas including internal capsule and corpus callosum appeared weakly NGF as well as GDNF weakly positive.

20) The symmetrical lesions located in the genu and in the splenium of corpus callosum on MR and CT are major angiographic abnormalities.

21) The appearance diffusion coefficient and the fractional anisotropy were measured for the splenium of the corpus callosum, the genu of the corpus callosum, and the hippocampus.

22) Results: Cerebral white matter fibers including arcuate fibers, superior longitudinal fasciculus, internal capsule, superior fronto-occipital fasciculus and corpus callosum were involved.

23) Additionally, high ADC values were found in the hippocampus callosum, which connects the two cerebral hemispheres.

24) Objective : To evaluate the CT and diagnostic value of the corpus callosum injury.

25) Tissue, Array, Human Adult Normal, Brain II, Brain, Frontal lobe, Temporal lobe, Parietal lobe, Occipital lobe, Pons, Thalamus, Corpus callosum (Paraffin.

26) Cerebellar hypoplasia is not a sonographic feature of isolated agenesis of the corpus callosum.

27) Conclusion: We could not identify a single etiologic factor responsible for the lesion in the splenium of the corpus callosum.