1. In both patients naloxone led to great improvement, supporting the idea that endogenous opiates are involved in the condition.

2. Objective:To investigate the therapeutic effect of naloxone on ischemic hypoxic cerebropathy in neonates.

3. New application development of Naloxone in emergency and critical disease was discussed.

4. Conclu. sion : Naloxone can protect or improve the immunologic function in aluminium - poisoned mice.

5. In addition, the withdrawal symptoms precipitated by naloxone in morphine dependent rats were blocked by methoctramine(ip) or pirenzepine (it) at single dose injection in a dose dependent manner.

6. Conclusion High-dose Naloxone treatment on intoxation of hypnotic and alcohol is more effective than low-dose.

7. Objective To investigate the effect of naloxone on neurologic protection after brain trauma.

8. AIM : To evaluate the instant effect of naloxone for brain infarction with somatosensory evoked potentials ( SEP ).

9. To evaluate the instant effect of naloxone for brain infarction with somatosensory evoked potentials ( SEP ).

10. AIM: To observe the efficacy of naloxone treatment of primary brain-stem injury.

11. Methods:Summarizing the usage of naloxone in acute intoxation, acute cerebral infarction, acute respiratory failure, critical craniocerebral injury and shock.

12. Conclusion Naloxone treatment of joint meclofenoxate good effect in severe brain injury, it is worth promoting in clinical use.

13. In particular, those injected with naloxone did no better than the o ther two control groups.

14. AIM: To investigate the effects of tanshinone and naloxone on regional myocardial blood flow (RMBF) during ischemia and reperfusion.

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15. Objective To explore the role of naloxone in treating vertebral - basilar artery blood insufficiency.

16. Objective To observe the effectiveness of naloxone treatment for acute alcohol toxicosis.

17. To explore the influence of Naloxone Hydrochloride injection on T - AOC in acute alcoholism rats.

18. Objective : To study the application of naloxone in acute and critical diseases.

19. In such cases, drugs such as naloxone and naltrexone provide molecules that bind to opioid receptors with a higher affinity than the opioids themselves, but do not activate the receptors.

20. Objective To investigate the effect of naloxone in the treatment of acute organophosphorous pesticide poisoning.

21. Conclusion:Naloxone can improve the conditions of ischemic hypoxic cerebropathy in neonates to a definite degree and facilitates recovery.

22. Objective : To observe the curative effects of naloxone therapy on primary apnea of premature infants.

23. It has been observed in rabbits that hypotension caused by haemorrhagic orendotoxic shock could be reversed by injecting naloxone, phentolamine or cinanserineinto the lateral cerebroventricle.

24. Objective: To explore the leu-enkephalin(L-ENK) levels in the plasma and CSF of patients with cerebral hemorrhage, and the effect of naloxone on the levels.

25. Objective: Observe the curative effect of acute intensive alcohol poisoning by using naloxone hydrochloride.

26. Moreover, the potential analgesic mechanism of cinobufagin injection was investigated by injecting naloxone hydrochloride into mice.

27. Methods:The firing of ventral pallidal neurons in the action of morphine and naloxone was recorded and analyzed in the morphine dependent rats with extracellular single-unit recording.

28. The study suggested that elevated levels of endogenous opiate peptides in blood and CSF were involved in pathogenesis of epidemic encephalitis-B and naloxone is an effective therapy.

29. Conclusion: To prevent the toxic encephalopathy must actively shock and use Naloxone to clean raw surface.

30. However, the analgesic effects of acupuncture and morphine were not reversed by naloxone microinjected into the amygdala or the mesencephalic reticular formation. 2 .

31. The key of naloxone therapy is early large dose naloxone intravenous injection.

32. AIM: To evaluate the efficiency of naloxone in treating newborn infant respiratory insufficiency.

33. Objective To observe the clinical effect and mechanism of puerarin and naloxone in treatment for concussion.

34. Aim To investigate the changes of dynorphin and enkephalin with naloxone in rat's brain tissue and the pain threshold of tail flick after positive and negative acceleration loaded.

35. As an apiod receptor antagonist, naloxone used to be a special antidote of meconiums.

36. A simple and sensitive HPLC-electrochemical detection method has been developed for the determination of naloxone hydrochloride and naltrexone hydrochloride in plasma.

37. The firing of ventral pallidal neurons was enhanced prominentlyin addictive rats, and it could be reversed by naloxone completely.

38. Objective To observe the clinical effect of naloxone hydrochloride on the treatment of acute cerebral infarction.

39. Objective To assess the curative effect of hepatic cerebropathy with naloxone.

40. Naloxone is a narcotic antagonist that prevents or reverses the effects of opioids. It is widely used in the treatment of narcotics and alcohol intoxication, shock, cerebral infarct.

41. The effect of naloxone is mainly to increase neuronal excitability at thetimeto ameliorate nervous conductivity.

42. Objective To investigate the effects of microinjection of morphine and naloxone into basolateral amygdaloid nucleus on sleep of rat.

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43. Objective To observe the efficacy of naloxone in the treatment of acute alcoholism.

44. Ano ther received the same regime, but the saline was combined with naloxone, an opiate - blocking drug.

45. The synthesis of naloxone was improved and the method for O - demethylation was investigated.

46. Objective To evaluate the effect of different dosage of Naloxone treatment on intoxation of hypnotic and alcohol.

47. Objective : To discuss the value of using large dosage naloxone hydrochloride to salve acute alcoholism patients.

48. Objective : To evaluate the effects of naloxone ( NX ) on cirrhotic patients with subclinical hepatic encephalopathy ( SHE ).

49. Objective To study the expression of interleukin 18 (IL-18) and the interfering effects of naloxone in the brain edema induced by lipopolysaccharide (LPS) in rats.