1. Several hormones may stimulate platelet activating factor biosynthesis invitro.

2. These results suggest the involvement of platelet activating factor in the endothelin induced fibrinolytic activation and subsequently developed mucosal haemorrhagic lesion.

3. Therefore the platelet-aggregate ratio would be low in the presence of circulating platelet aggregates.

4. The possibility that intestinal epithelial cells may produce platelet activating factor has been suggested by Kald etal.

5. The content of platelet activating factor in pancreatic tissue was determined using bioassay technique with washed rabbit platelets as described previously.

6. Occasionally, a substantially increased platelet or white cell count may lead to apparent increases in plasma potassium concentration.

7. As knowledge of basic platelet metabolism increases it will be more easy to identify platelet abnormalities and their pathogenesis in diabetes mellitus.

8. This hypothesis deserves further study as platelet activating factor metabolism in the stomach lumen is unknown.

9. These authors have shown that colonic platelet activating factor content was significantly increased irrespective of colonic inflammation.

10. Lyso platelet activating factor and alkyl acyl glycerophosphocholine were not significantly higher in Crohn's disease than in controls.

11. This led to a substantial increase in platelet cholesterol concentration, the bulk of this increase being associated with the platelet membrane.

12. Biological activity of platelet activating factor was measured by platelet aggregation using a Chrono-log Corporation aggregometer.

13. The mechanisms by which platelet sticks to platelet during aggregation are not fully understood.

14. Platelet activating factor also caused a marked decrease in the pancreatic blood flow comparable with that observed after caerulein infusion.

15. Formalin fixes platelet aggregates which would be removed during the centrifugation to produce platelet-rich plasma.

16. Platelet-adhesion studies Early studies of platelet function in diabetes used in vitro tests of platelet adhesion.

17. Other authors found similar findings with regard to circulating platelet aggregates.

18. In addition, improved therapeutic agents with more specific and controlled effects on platelet metabolism may be developed allowing more effective intervention.

19. There are, however(), some differences in the action of caerulein and platelet activating factor on the pancreas.

20. According to our finding caerulein infused at supramaximal dose increased the production of platelet activating factor in the pancreatic tissue by about nine-fold.

21. The actin filaments extend into the cytoplasm from this point and form long filaments orientated concentrically around the platelet granules.

22. However, bearing in mind these constraints; what is the current evidence linking diabetic control and platelet function?

23. The above studies have attempted to assess the presence of circulating platelet aggregates.

24. However, this suggestion depends on the assumption that prostacyclin is continually produced by arterial wall to prevent platelet aggregation.

25. Platelets are studied in an artificial environment and the very process of preparing the platelet sample for study may affect their behaviour.

26. About 24 hours before assay the sample was placed in 80% ethanol to extract platelet activating factor from the tissue.

27. For these reasons studies detailing the effects of diabetic control on platelet function must be interpreted with caution.

28. This has led to difficulties in interpretation of the possible sequence of events in the development of enhanced platelet reactivity in diabetics.

29. These findings suggest that lipoprotein abnormalities in the diabetic state may also indirectly affect platelet function by damaging vascular endothelium.

30. Gliclazide is the sulphonylurea that has received most study in terms of possible effects on platelet function.

31. Local application of platelet concentrates shows potential in treating diffuse mucosal haemorrhage due to thrombocytopenia.

32. The significance of platelet activating factor in the gastric juice of healthy humans is still difficult to analyse.

33. The role of platelet activating factor blocker in the treatment of inflammatory bowel disease deserves investigation.

34. These findings have led to the suggestion that altered endothelial function without endothelial loss may be important in permitting platelet thrombus formation.

35. The L-arginine-NO system exerts various biological actions including vascular smooth muscle relaxation and inhibition of platelet aggregation.

36. Spontaneous platelet aggregation Several studies have attempted to assess whether there is evidence of increased circulating platelet aggregates in diabetic subjects.

37. A positive correlation was found between glycosylated haemoglobin concentration and the prostacyclin concentration necessary to inhibit ADP-induced platelet aggregation by 50 percent.

38. Studies in this field are difficult to interpret due to the inherent variability of the measurements of platelet function.

39. Thus diminution of prostacyclin production and stimulation of platelet aggregation both mediated by lipid peroxides could contribute to thrombosis on atheromatous plaques.

40. In addition we have also investigated the involvement of platelet activating factor in the endothelin induced fibrinolytic activation.

41. Platelet activating factor was extracted from biopsies using ethanolic extraction as previously reported by us and others.

42. Platelet binding falls after the menopause and rises in women using the Pill.

43. The mechanism of lipid-induced effects of platelet function has received considerable attention.

44. When the platelet count in your blood drops too low, your blood does not clot as well as usual.

45. We found a relation between platelet activating factor precursors and acid, and between platelet activating factor precursors and pepsin.

46. Therefore we suggest that platelet activating factor should be considered as a global mucosal activity marker.

47. Lipoproteins may indirectly affect platelet function by altering vascular endothelial cell function.

48. In addition to problems intrinsic to the methodology of platelet function(), problems also arise from the diabetic groups studied.

49. Inhibitors of prostaglandins also inhibit the increased platelet aggregation, which is present in the early phases of migraine.

50. Both cell types generated platelet activating factor activity and this was generally comparable for epithelial and lamina propria cells.

51. Along with this there was a significant reduction in circulating platelet aggregates.

52. This would enable prospective studies to be performed to determine the importance of platelet function in the development of vascular disease.

53. We can not exclude the possibility, however, of abnormal metabolism of platelet activating factor in four of them.

54. This correlation argues for the possibility of synthesis of platelet activating factor precursors in cells sensitive to gastrin stimulation.

55. In this study, we found platelet activating factor in four of 13 normal subjects.

56. Platelets were resuspended in washing buffer at appropriate concentrations for platelet aggregation measurement.

57. They are known to counteract both the vasoconstriction and the enhanced platelet aggregation present in the initial phase of migraine.

58. Although platelet thromboxane generation was elevated in diabetics without clinical evidence of vascular disease(Sentence dictionary), the difference did not reach statistical significance.

59. Maternity blues linked to platelet receptors TWO-THIRDS of women suffer from postnatal depression, accompanied by crying, confusion and tension.

60. The level of platelet activating factor in the rat gastric mucosa was determined after the administration of endothelin-1 using a radioimmunoassay method.

61. For instance, there is no absolutely satisfactory test of platelet function.

62. Correlation with platelet count was particularly good in colonic disease.

63. Platelet activating factor may be involved in the pathogenesis of ulcerative colitis as well as in experimental colitis.

64. Platelet activating factor causes gastrointestinal mucosal injury and ischemic bowel necrosis.

65. This latter finding provides further evidence of the in vivo platelet activation and release reaction.

66. These results indicate that platelet activating factor may be an important factor in the progression of acute pancreatitis provoked by caerulein.

67. The earliest finding is the platelet adhesion to collagen fibres followed by aggregation and formation of a platelet plug.

68. Aggregating activity of the samples was measured over the linear portion of the calibration curve obtained with synthetic platelet activating factor.

69. Very low platelet count may lead to severe Bleeding.

70. Inhibits platelet aggregation. DNA polymerase inhibitor. Shows antifungal activity.

71. Here is an actual electron micrograph of a platelet.

72. Ticlopidine is a new platelet aggregation inhibitor.

73. White cells contaminate the red cell and platelet components.

74. Objective To study the clinical value of platelet parameters in diagnosis of idiopathic thrombocytopenic purpura.

75. Conclusion:Longstand possesses the effects of endotheliocyte protection and platelet function improvement.

76. Conclusion: The expression of P - Selectin seems to be correlated with endothelial cell injury, platelet and thrombosis.

77. Myelodysplasia, which is associated with low platelet counts, predisposes some patients to leukemia.

78. Conclusions Hip prosthesis can cause intravasation of bone marrow fat that leads to platelet activation.

79. Using the recombinant protein, several investigators have shown that thrombopoietin influences all aspects of megakaryocyte development, from the hematopoietic stem cell to the mature platelet.

80. Conclusion:The results suggest that there is a prethrombotic state in patients with CHD, manifested as impaired fibrinolysis , vascular endothelin cells disfunction and platelet activation.

81. Objective To study the relationship between platelet - derived growth factor ( PDGF ) and hilar cholangiocarcinoma ( HCCA ).

82. In addition, the role of anti-allergic, anti - platelet aggregation and inhibition of MMP synthesis.

83. ADP - P 2 Y 12 receptor pathway plays a crucial role in platelet activation and thrombogenesis.

84. Aim To investigate the effects of mailuoning oral liquid on platelet aggregation, thrombosis and fibrinolysis.

85. Objective:To explore effects of Xiaoshuan Jiangzhi Pellet(XSJZ) on platelet activation functions and plasm fibrinolysin activities in patients with transient ischemic attack(TIA).

86. Objective:To study the effect of sodium ferulate on the abnormal function of platelet in patients with ulcerative colitis(UC).

87. Thrombopoietin (TPO) is a novel cytokine that is involved in the regulation of platelet production.

87. is a online sentence dictionary, on which you can find excellent sentences for a large number of words.

88. It was resulted that isoflavone aglycones inhibited human platelet aggregation induced by ADP, collagen and epinephrine.

89. Commonly believed by scholars is that five substances play important roles in the formation of AS, i. e. vessel smooth muscle cell, Vessel endothelium cell, platelet, lipoprotein and monocyte.

90. AIM: To investigate the platelet aggregations in patients with coronary heart disease(CHD) and the effect of ticlopidine treatment.

91. Conclusion An inhibitor of platelet aggregation from I. sinensis was identified, which may play an important role for ticks to successfully get blood meal from their hosts.

92. With the same method, activity of coagulation factor II and platelet aggregation rate were furthermore measured in rats.

93. Objective: To study the relationship between hyperviscosity syndrome (HVS) and platelet membrane glycoproteins (GP) sub-group changes.

94. Certain enzyme studies, like the white cell or platelet assay, are particularly advantageous when testing in pregnancy.

95. Methods Platelet count in 112 patents with lung cancer was analyzed.

96. Evaluates the performance for COULTER STKS fully automatic haemacytometer analyzer to assay blood platelet.

97. Objective To observe the curative effect of deproteinized xenogeneic bone(DXB)with platelet rich plasma(PRP)to repair bone defect of femur.

98. Objective To study the action of ticlopidine in inhibiting EDTA-dependent platelet aggregation in vitro.

99. Objective : To established HPA genotyping method and explored platelet transfusion refractoriness.

100. Idiopathic thrombocytopenic purpura is the disease with participation of immunologic mechanism with decreased thrombocytopoiesis and increased platelet destruction.

101. Suboptimal smears had higher RBC count and hemoglobin level and lower platelet count in patients with myelodysplastic syndrome: refractory anemia.

102. Methods:Use the platelet function survey kit from CAP, perform the platelet function test according to routine laboratory procedure.

103. The present invention discloses the application of gingerol oxime in preparing platelet aggregation inhibitor.

104. Objective To study the clinical significance of platelet membrane glycoproteins in patients with SIRS and MODS.

105. Objective To observe the effect of naftopidil ramification - BWYJ, on the platelet aggregation of rabbit.

106. Platelet production begins with invagination of the megakaryocyte cell membrane and the formation of cytoplasmic channels and islands.

107. Methods: The changes of blood viscosity, haematocrit, platelet aggregation and fibrinolytic activity the treatment with XDGI.

108. The lower the HU, the more platelet - rich was the thrombus composition.

109. Objective To assess the effect of cardiopulmonary bypass(CPB) on platelet in patients with cyanotic congenital heart disease.

110. Platelet activation by P-selectin and monocyte cell surface P-selectin glycoprotein ligand- 1 combine to form PMA.

111. Objective To investigate the Clinical significance and the changes of serum parathormone (PTH) in the platelet donors with long-term donation.

112. Objective To study the relationship of serum thrombopoeitin (TPO) level and peripheral blood platelet count in patient with acute trauma and its clinical significance.

113. Aspirin resistance can be detected by laboratory tests of platelet thromboxane A2 production or platelet function that depend on platelet thromboxane production.

114. And the rat model of thrombosis in vivo was established to study aggregation of blood platelet.

115. Measurement of serum (or plasma) thromboxane B2 is labour intensive, not readily available, and might not be specific for platelet function.

116. Objective To explore thrombocytosis in patients with colorectal cancer and the interaction of platelet and tumor metastasis.

117. TTD, an effective formula for treating UC,(http:///platelet.html) can improve the pathological injury in the colon of the UC rats and inhibit the hyperfunction of blood platelet.

118. Objective : To determine the acid composition and isoelectric point of porcine platelet - derived growth factor.

119. There are cruor system, fibrinolysis system and blood platelet system which affect thrombosis.

120. The temperature of platelet intracellular ice crystal formation (IIF) is one of the most important physical parameters to instruct platelet cryopreservation.

121. AIM: To study the effect of sphingosine 1-phosphate (S1P) on the increase in microvessel permeability induced by platelet activating factor (PAF).

122. Platelet function was performed at three time points: at baseline, 14 days after randomization, and 14 days after treatment cross-over.

123. Aim To study the Effects of different extracts from Bletilla colloid on rabbit platelet aggregation.

124. "If there was a suitable platelet substitute, that would be a tremendous achievement for clinical medicine, " says hematologist Marcel Levi of the University of Amsterdam in the Netherlands.

125. Aspirin resistance is the inability of aspirin to reduce platelet production of thromboxane A2 and thereby platelet activation and aggregation.

126. COX-1 regulates the homeostatic or housekeeping activities in the body, such as platelet aggregation, gastric acid secretion, and renal function.

127. Objective To investigate the effect of Jiawei Shaoyao Decoction on the function of platelet in patients with chronic ulcerous colonitis of Damp-Heat syndrome.

128. Objective To discuss the clinical application of therapeutic platelet apheresis in the high platelet patients and to observe it's impact on platelet function.

129. AIM: To investigate the effects of MK-447 on platelet shape change, aggregation, and ATP release by collagen (Col), ADP, and stable analogue of thromboxane A2 ( STA2 ) in rabbits.

130. Note that this platelet bears a striking resemblance to a chocolate chip cookie.

131. Conclusion Insulin can inhibit P - slectin expression on activated platelet membrane.

132. The platelet abhe - sion and aggregation could be recovered after washing and removing DMSO.

133. Aspirin resistance can be overcome by treating the cause or causes, and reduced by minimising thromboxane production and activity, and blocking other pathways of platelet activation.

134. Platelet activation is thought to be an important risk factor in blood clotting.

135. These Japanese Islands, on the southern tip of Japan, are actually on the Okinawa platelet, which would lose support as the Philippine Plate tips. Note the date, June 12, 2011.

136. In this study, we have observed the effects of extracting from Carthamus tinctorium L on blood coagulation, platelet aggregation and thrombosis.

137. Objective To assess the influence of apheresis and wash on platelet function and morphology.

138. The physiopathology characteristic of pulmonary heart disease is that the vessels of the lungs are tic, resistance of pulmonary circulation is increased, the blood platelet is gathered, et al.

139. Conclusion The parameter of platelet are important consult of judgment malady, injury of heart, liver nephridium and platelet count was correlation.

140. Von Willebrand disease is characterized primarily by a defect in platelet adhesion to the endothelium.

141. EGB have definite antiplatelet aggregative activity, whether is it also through effecting the activity of platelet PDE to inhibit platelet aggregation?

142. Platelet derived growth factor is FDA-approved for treatment of diabetic ulcers and to promote bone repair in tooth-associated defects, but time-release delivery has been a big problem.

143. Conclusion The ESW's impacting is able to damage the platelet, and result in coagula...

144. Objective To explore the influence of routine storage or cryopreservation on platelet function.

145. Objective To explore the relationship between platelet transfusion and LCT in hematologic malignancy patients.

146. In order to evaluate the blood compatibility of these membranes, we did a series of test, such as SEM observation of the platelet adhesion to membrane surface, blood clotting and haemolysis ratio.

147. It was also shown thai ticlopidine could significantly inhibit platelet TXB2 formation stimulated by adenosine diphosphate(ADP) .

147. is a online sentence dictionary, on which you can find excellent sentences for a large number of words.

148. The granulosa increasingly disintegrated and the yolk platelet came into being in the oocyte in vitellogenesis .

149. After cultured for 1 week, the tissue engineered heart valve and unseeded scaffold were tested by platelet adhesion experiments.

150. The splenectomy also removes a suppressant of bone marrow platelet production or release.

151. Nitric oxide (NO) can inhibit platelet adhesion, aggregation, and activation as well as relax smooth muscles and modulate neurotransmission.

152. COX-1 is widespread in many tissue including to blood vessel, stomach, kidney and the platelet in charge of signal transmission and the maintenance of cell function's balance.

153. Results:Both quercetin monosulfate and 5' chloro 5' deoxyadenosine had inhibitory effects on platelet aggregation, the combination of them enhanced those effects.

154. Methods: We compared the changes of blood viscosity, haematocrit, platelet aggregation and fibrinolytic activity after the treatment with XDGI.

155. The platelet adhesion on surface of fibrillar collagen under high shear rate condition was observed by parallel plate flow chamber with McAb.

156. The changes of pro and posthemodialysis in platelet counting, thrombin time and activated partial thromboplastin time were investigated.

157. Conclusion:Quercetin monosulfate in combination with 5' chloro 5' deoxyadenosine had synergetically inhibitory effects on platelet aggregation.

158. The method is similar to light or optical aggregometry except that it can be done in whole blood, thus obviating the need for preparation of a platelet suspension.

159. Objective To investigate the platelet inhibition ratio by thromboelastography (TEG) and its clinical impact in patients undergoing percutaneous coronary intervention (PCI).

160. Prothrombin time was prolonged and ADP - induced platelet aggregation was inhibited in middle and high dose group.

161. Results:Storax cultivated in Guangdong could inhibit thrombosis in vivo, reduce blood viscosity and hematocrit, and decrease platelet aggregation.

162. OBJECTIVE To observe the effects of phenolic alkaloids of menispermum dauricum(PAMD) on platelet aggregation and explore its mechanism.

163. Method Platelet aggregation was induced by adenosine diphosphate ( ADP ) in rabbits.

164. Objective:The primary blood platelet reduction hemacelinosis is one kind of common hemorrhage disease.

165. Objective To assess the influence of apheresis and wash on platelet function and morphology. Washed platelet of quality and clinical effect were observed.

166. The rate of platelet aggregation induced by ADP, COL, AA was determined.

167. Defibrase can effectively reduce blood viscosity, content of fibrinogen, hemocyte aggregation and platelet aggregation.

168. Objective To observe the effect of Protein C Activator(PCA) from Venom Agkistrodon halys Pallas on platelet aggregation in rats with septicaemia .

169. Objective: To investigate the thrombolysis effect of shuanglong staphylokinase on the cerebral platelet rich microthrombi in vivo.

170. Low platelet counts presents a common but an especially vexing problem.

171. Objective:To study the effects of atorvastatin on platelet activity, blood lipids in diabetic patients with hyperlipidaemia.

172. Can the blood platelet use red bean to treat coming China fir?

173. Conclusion The frozen platelet has got the positive impact to the hemostasis.

174. High platelet levels do not necessarily signal any clinical problems, and are picked up on a routine full blood count.

175. Styrax produced in Guangdong could reduce blood viscosity and hematocrit, and decrease platelet aggregation.

176. Albumin, oxygenation index, myocardial enzyme, body temperature of the admission day, maximum body temperature in hospital, platelet may suggest of septic shock.

177. Objective To explore the platelet activation state in patients with acute coronary syndrom(ACS) and its significance.

178. Halloysite has marked inhibition on the ADP-induced platelet aggregation both in vitro and half in vivo,(http:///platelet.html) and has obvious antagonism on the thrombosis in mice induced by ADP.

179. Objective: To investigate the effects of scutellarein on thrombosis, platelet aggregation and hemorheology in rats.

180. Methods:Forty-six UAP patients and 46 healthy subjects were evaluated with 6 psychological scales, and their adenosine diphosphate(ADP)-induced platelet aggregation function was determined.

181. Objective blood platelet of the patients with cerebral infarction to study the therapy method.

182. Objective : To explore the preparative method for blood platelet concentrate under the aseptic condition.

183. Conclusion Naokang can dose - dependently inhibited the platelet agglutination.

184. Methods: The platelet aggregation rate was measured by turbidimetric method.

185. Conclusion The treatment aimed at the blood platelet may slow down the development of DN.

186. Albumin, oxygenation index, myocardial enzyme, body temperature of the admission day, maximum body temperature in hospital, platelet may suggest of death.

187. Murphy MF, Waters AH . Immunological aspacts of platelet transfu sions. British Journal of Haematology 1985, 60:409.

188. Conclusion: The activated small volume platelet is a character of UC.

189. They injected some fluorescent dye to watch what happened, and during 72 hours of filming, they watched their platelet plastic grow a series of tubules.

190. Objective:To discuss the reasons of false decreasing in platelet count by automated blood cell counter, to provide evidences on clinic diagnose and cure idiopathic thrombocytopenic purpura.

191. I would also like to highlight some other trials based on their use of new anti-coagulant and anti platelet agents either in the setting of acute coronary syndrome or PCI including the AVERROES Trial.

192. Arteries were distended and occluded by necrotic intraluminal debris, without platelet fibrin thrombi.

193. Methods 1. The platelet aggregation induced by ADP(Adenosine diphosphate) and collagen were established in vivo and in vitro and the effect of EMS on platelet aggregation was observed.

194. To inquire into the relation between cancer and blood platelet count ( BPC ).

195. Objective to observe relationship among TCM syndrome differentiation, hemorrheological and platelet activating factor in the podagra arthritis of acute of episode.

196. Conclusion Personal film intervention can alleviate anxiety and ache significantly for apheresis platelet donors and have a positive influence on blood pressure and heart rate.

197. Conclusion In this group, the higher the LCT titer, the more likely the ineffective platelet transfusion.

198. Objective To observe the inhibition effects of Naokang on platelet agglutination.

199. Conclusion It is suggested that LCT positive is mainly immune factor for ineffective platelet transfusion.

200. The wavelength converting element 110 may be, e. g. , a phosphor platelet that is indexed matched with the optical concentrator 108.

201. Results The isotype platelet antibody was 46.72% in repeated transfusion and concentrated platelet transfusion.

202. Platelet aggregation was measured turbimetrically and plasma fibrinogen level was determined by biuret method.

203. Aim To observe the effect of phenolic alkaloids of Menispermum dauricum (PAMD) on thrombosis and platelet aggregation, and to explore its mechanism of action.

204. Objective:To study the changes and clinical significance of the megakaryocyte and the blood platelet parameters of patients with malnourished megaloblastic anemia.

205. Objective: To observe the effect of Xiaoyu Zhixue tablet (XYZXT) on expression of platelet membrane glycoproteins in patients with hemorrhagic thrombopathy, and to explore its possible mechanism.

206. Objective:To study the variety and value of large platelet in peripheric blood of pregnant women .

207. Haemolytic uraemic syndrome (HUS) is a life-threatening disease characterized by acute renal failure (uraemia), haemolytic anaemia, and a low platelet count (thrombocytopenia).

207. try its best to gather and build good sentences.

208. Results: LTPI has significantly prolonged the clotting time and decreased the platelet aggregation function in vitro.

209. Objective To explore the clinical significance of platelet, prothrombin time , activated partial thromboplastin time , thrombin time , plasma fibrinogen for parturient women.

210. There were no significant intergroup differences in lymphocyte and platelet counts at all time points observed.

211. The adenosine diphosphate (ADP)-stimulated platelet aggregation (PA) was assessed by conventional aggregometry.

212. NSA had no effect on the platelet aggregation induced by AA.

213. Function of platelet; Anticoagulation system; fibrinolysis; The principles of blood transfusion.

214. The term of "aspirin resistance" usually refers to laboratory resistance. It can be identified by measuring thromboxane A2 or thromboxane-dependent platelet function.

215. Adenosine diphosphate ( ADP) was analyzed by reversed phase HPLC, and rabbit platelet aggregation was measured turbimetrically.

216. Platelet activation has a closed relationship with thrombogenesis in Stroke patients.

217. The activation of random donor platelet concentrates and platelets apheresis collections ( plateletpheresis ) during storage were studied.

218. The main adverse reactions were hematologic toxicity. The manifestations were : WBC, neutrophile granulocyte, blood platelet and hemoglobin all decreased, then was digestive canal symptoms.

219. Changes of platelet count (PC) and mean platelet volume (MPV) were observed in anesthetized open-chest dogs after coronary occlusion.

220. Activated platelets are the primary source of LPA in circulation, which in turn stimulate platelet aggregation.

221. CBC with platelet count, fibrinogen level, partial thromboplastin time, and ABO blood typing and antibody screen are obtained.

222. The blood coagulative system in three groups was evaluated by testing platelet count (PLC), Active part thrombin time (APTT), prothrombin time (PT), thrombin time (TT), FIBrinogen(FIB).

223. Objective : To discuss the influence and mechanism of action on platelet agglutinate - tion by Qidantian capsule.

224. Objective : To improve the method for isolating platelet - rich plasma ( PRP ).

225. Objective: To determine the amino acid composition and isoelectric point of porcine platelet - derived growth factor.

226. The D-dimer, fibrinogen, hematin, and blood platelet were measured in these patients before and after urokinase administration.

227. Splenectomy and newer agents to boost platelet production are also under study in chronic ITP.

228. Objective To observe the expression change of platelet membrane glycoproteins of patients with hemorrhagic thrombopathy, and to explore its possible pathogenesis.

229. Thrombocytosis is often associated with chronic blood loss and iron deficiency because of increased platelet production in the marrow reacting to continued consumption and loss.

230. Methods Recalcification test, hemolysis test and platelet adhesive test were conducted for compact silk membrane, polyporous silk membrane, bovine pericardium and collagen sponge.

231. Methods:Count the platelet by automated blood cell counter in vein blood and diluted fingertip blood and smear of blood check by microscope.

232. Probably the most common platelet function defect is the irreversible inhibition of thromboxane (which is necessary for platelet aggregation) caused by aspirin administration.

233. It is suggested that the platelet reaction can serve as a marker of agonal stab wound and the leucocyte reaction as a criterion for timing of wounds is not feasible.

234. Platelet disorders are either quantitative (thrombocytopenia or thrombocytosis) or qualitative (thrombocytopathy).

235. The results of activity analysis suggested that these two chimera proteins not only have antithrombin activities, but gain platelet aggregation inhibitory activities as well.

236. FG is prepared from platelet - rich plasma or by mixing concentrated fibrinogen solutions with thrombin.

237. Prostaglandin is the dilation of blood vessels and inhibit platelet function.

237. try its best to collect and build good sentences.

238. Objective To investigate the effects of personal film intervention in the apheresis platelet donors on anxiety and pain caused by vein phlebotomy during donating.

239. AIM: To compare the efficacy and safety of clopidogrel on anti platelet aggregation with ticlopidine.

240. Results Naokang can inhibited the platelet agglutination induced by ADP and AA.

241. Objective To study the change of platelet activation and enhanced coagulability in patients with chronic pulmonary heart disease (COPD).

242. It is simply a matter of decreasing sequestration and destruction of platelet.

243. This study investigated 112 patients with bone marrow failure needing repeated platelet transfusion. The incidence of induced alloantibody was 53. 6%.

244. The Separation efficiency of red blood cell(), white corpuscle and blood platelet from pig blood was investigated by chromatographic columns of four celluloses and two ion-exchange resins.

245. Objective To observe the levels of platelet alpha-granule membrane glycoprotein 140 (GMP-140) and fibrinopeptide A (FPA) in patients with acute coronary syndromes (ACS).

246. Inhibition of early steps of platelet adhesion to the ischemic endothelium and the subendothelial matrix may offer a novel and safe treatment strategy in acute stroke.

247. It is concluded that the occurrence of abnormal platelet aggregative function is high in patients with MPD.

248. Conclusion:Garlicin could reduce the brain injury in rats with focal cerebral ischemia, and the possible me ch-anism is to inhibit the activation of platelet.

249. CONCLUSION: The platelet aggregations were increased in patients with coronary heart disease and could be inhibited by ticlopidine.

250. Conclusion FCM assay can be used as a rapid and sensitive method for detecting platelet-associated antibody and platelet crossmatching.

251. Conclusion:XSJZ can decline platelet activation functions and improve plasma fibrinolysin activities and takes on longer effect to TIA patients, so it can decrease or delay the recurrence rate of TIA.

252. Canadian Blood Services' plasma donors may undergo apheresis up to 52 times a year while platelet donors may donate 26 times a year.

253. Objective To study the relationship between microangiopathy and activated platelet in patients with - insulin - dependent diabetes mellitus ( NIDDM ) .

254. Methods Recalcification, hemolysis and platelet adhesive test were conducted for the compact silk membrane, porous silk membrane, bovine pericardium and collagen sponge.

255. Further studies suggested that accurate measurements could be made at platelet concentrations up to 4.8 million per microliter.

256. Objective To assess diagnostic value of thrombocytosis through observing platelet count in patients with lung cancer.

257. This might be explained by altered resorption, altered lier metabolism, or altered platelet response.

258. The patients with antibody produced after many platelet transfusions were transfused with CDC matched platelet.

259. Results:After Longstand treatment, the vascular endotheliocyte and platelet functions were markedly improved in addition to improvement in neurological function.

260. The platelet count remained low with no clinical change until pentoxifylline was started at the age of 15 months.

261. Objective To investigate the clinical efficacy of matched apheresis platelet by cross matching test.