1. As expected, graft atherosclerosis becomes problematical in long term survivors of cardiac transplantation.

2. They may be due to senile atherosclerosis.

3. Objective:To study the mechanism of pravastatin enhancing atherosclerosis regression.

4. The compound ion salt retarded atherosclerosis.

5. Atherosclerosis ( AS ) is an inflammatory disease.

6. Elastic vessels also resist the hardening activity of atherosclerosis.

7. Atherosclerosis is the major pathological basis of the cardio- cerebrovascular diseases. Its occurrence and development is closely associated with oxidative lesion and abnormality of lipid metabolism.

8. The severe atherosclerosis in the aorta at the top shows extensive ulceration in the plaques.

9. Vascular calcification is a common phenomena among atherosclerosis, diabetes, chronic kidney failure and aging.

10. Conclusion: Probucol may stabilize or decrease carotid atherosclerosis plaque, which is of great clinical significance for prevention and treatment of coronary heart disease.

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11. Results: Except the CTRL group, lipide stripe and atherosclerosis plaque were found in other groups.

12. Aortas with mild , moderate , and severe atherosclerosis, gross.

13. It is often claimed that high blood cholesterol levels promote atherosclerosis and consequent coronary heart disease.

14. Isner and his colleagues studied 10 patients with severe atherosclerosis.

15. So the endothelins may have a major role in atherosclerosis and the reocclusion that often follows surgical opening of occluded arteries.

16. Dr. Asa Tivesten and colleagues at Goteborg University prospectively followed 313 white men enrolled in the Atherosclerosis and Insulin Resistance Study.

17. In conclusion, ABI measured using the automated oscillometric method can be used to predict the severity of coronary atherosclerosis in patients with CAD.

18. More and more data suggest that the potential role of infection develop atherosclerosis and the injury of vassal dissepiment is one of main cause of atherosclerosis.

19. Serological evidence of infections and Type 2 diabetes: the MultiEthnic Study of Atherosclerosis.

20. Background — enous bypass grafts may fail because of deelopment of intimal hyperplasia and accelerated atherosclerosis.

21. Objective: To explore the effect of Probucol on carotid atherosclerosis plaque.

22. Three aortas are shown to demonstrate mild, moderate, and severe atherosclerosis from bottom to top.

23. To observe the role of serum concentration of dehydroepiandrosterone (DHEAS) in male patients with coronary atherosclerosis.

24. Objective To study the levels of sex hormone and related receptors in elderly patients with atherosclerosis.

25. The liver-secreted protein Fetuin-A is elevated in insulin resistance, is an independent predictor of type 2 diabetes and is associated with atherosclerosis.

26. Most of the etiological factors are sudden cardiac death and sudden cerebral death, and coronary atherosclerosis is one of the main causes for exercise-related sudden death.

27. These three aortas demonstrate mild , moderate , and severe atherosclerosis from bottom to top.

28. Methods:To observe the influence of Pravastatin, Vitamin E and Captopril on the development of hyperlipidemia, atherosclerosis and hepatic steatosis in rabbits induced by high fat diet.

29. Background: The interaction of leukocytes and endothelium plays an important role in the development of atherosclerosis.

30. Objective To evaluate the relationship between vascular dementia ( VD ) and carotid artery atherosclerosis.

31. Methods The authors reviewed 42 patients underwent amputation for dry gangrene of leg due to atherosclerosis obliterans.

32. Disorders of lipid metabolism and atherosclerosis are common pathologic changes.

33. For hyperlipidemia, can be used for atherosclerosis, pressure, fatty liver of adjuvant therapy.

34. Progression of Subclinical Coronary Atherosclerosis: Does Obesity Make a Difference?

35. Changes of endothelial function and C - reaction protein ( CRP ) in hyperlipidemia and early atherosclerosis ( AS ).

36. AIM: To observe the effect of pravastatin on carotid artery atherosclerosis.

37. Objective To explore the clinical value of the abdominal aorta atherosclerosis spots observed with ultrasonography.

38. Background-The role of cholesteryl ester transfer protein in the development of atherosclerosis is still open to debate.

39. Measuring CIMT using ultrasonography is a well - accepted, noninvasive way to assess atherosclerosis.

40. And the typical atherosclerosis mottling in aorta lumen in model group under macrography and optical microscope were visible.

40. Wish you can benefit from our online sentence dictionary and make progress day by day!

41. Patients with hyperlipidemia, hypercholesterolemia, atherosclerosis and other cardiovascular and liver diseasesmust eat carefully.

42. The abnormality of serum lipid is the major risk factor of atherosclerosis and CHD, and it also increases the susceptivity of hypertension, obesity and insuline resistance.

43. Clinical phases of pathological state and treatment based on differentiation of signs of coronary atherosclerosis.

44. This article reviews the evidence and mechanism that human cytomegalovirus causes atherosclerosis.

45. Endothelial cell apoptosis plays a key role in the development of atherosclerosis.

46. Conclusion Pravastatin can decrease blood lipid dramatically, relieve atherosclerosis(), improve endothelium function and enhance the total treatment efficiency rate for angina.

47. The exact cause is unknown, but risks include high blood pressure, smoking, atherosclerosis (hardening of the arteries), trauma and Marfan's syndrome.

48. BackgroundPathologic studies in animals and human have clearly demonstrated that proliferation of adventitial vasa vasorum was associated with progression of coronary atherosclerosis.

49. Conclusion During the diagnosis of aortal atherosclerosis, the imaging velocity of B-TFE sequence is faster so that it is very valuable.

50. Atherosclerosis ( AS ) is a leading cause of cerebral infarction, especially the arterothrombtic cerebral infarction ( ACI ).

51. The creep curve is the relationship between changes in index, atherosclerosis of the ascending aorta has a certain impact on the creep characteristics.

52. Although the risk of losing a limb is low, hardening of the arteries in the legs is a marker for generalised atherosclerosis.

53. With sufficient funding, the focus on atherosclerosis would be broadened so that neurodegeneration, macular degeneration and lipofuscin accumulation would all also be targeted simultaneously.

54. Study on the Antioxidational Activity of Plasma on Experimental Atherosclerosis in Rabbits.

55. Recently, a preponderance of evidence supports thatinflammation plays an important role in a wide range of cardiovascular diseases, especially in the development of atherosclerosis.

56. Besides, diabetics , atherosclerosis patient , cholecystitis and pancreatitis patient do not drink the full-cream leben containing candy best , easy to aggravate state of illness otherwise.

57. The hemorheology indexes measured for 30 male patients with cerebral atherosclerosis in the paper.

58. Objective To investigate the utility of B-TFE sequence in the diagnosis of aortal atherosclerosis.

59. As one of proinflammatory cytokines, Interleukin - 18 ( IL 18 ) is highly expressed in atherosclerosis plaques.

60. Progression of symptomatic intracranial large artery atherosclerosis is associated with a proinflammatory state and impaired fibrinolysis.

61. Conclusion Monocyte proportion and erythrosedimentation are independent predictors of carotid atherosclerosis.

62. Studies substantiate that IMT correlates consanguineously with the presence of coronary atherosclerosis.

63. As atherosclerosis develops, the artery walls become thick and damaged and lose their normal inner lining.

64. Atherosclerosis is a multifactorial disease and inflammation mediates its all stages, from initiation to progression.

65. Objective To observe Tiaozhi capsules effect of protecting vascular endothelium, anti - atherosclerosis and discuss its mechanism.

66. Objective:This report is aimed to investigate the different effects of menopause on coronary atherosclerosis in patients with familial hypercholesterolemia and familial combined hyperlipidemia.

67. Objective To explore the correlation between hyperlipemia, hyperlipoproteinemia and unstable plaques of carotid atherosclerosis in ischemic cerebrovascular patients.

68. Background: Atherosclerosis is a chronic inflammatory - immune disease with enhanced oxidative stress.

69. Conclusion CMV plays an important role in the pathological process ofthe atherosclerosis and atherosclerotic cerebral infarction.

70. Objective:To observe the influence of Typha angustifolia L. on hyperlipidemia[/atherosclerosis.html], atherosclerosis(AS).

71. Objective To observe the effect of poplar skin monounsaturated fatty acid(PSMFA) on the lipid profile of atherosclerosis in rats.

72. Carotid ultrasound is a safe, noninvasive and relatively inexpensive means of assessing clinical and subclinical atherosclerosis.

73. To analyze the inflammatory mechanism of pulsatile low shear stress-induced atherosclerosis (AS) of the common carotid artery in rabbits in vivo.

74. Objective : To investigate the relationship between hyperglycemia and carotid atherosclerosis ( AS ) in Tibetan population.

75. Miller has been studying the link between atherosclerosis (hardening of the arteries) and nanoparticles which calcify within the arteries.

76. It can occur in atherosclerosis, diabetes mellitus, or decubitus ulcer, and after severe Burns or frostbite.

77. The pathogenesy of atherosclerosis is complicated extremely. There are several theories to explain it from different point of view.

78. Consequently, macrophage biology will be a major focus of the Keystone Symposia meeting on Molecular Basis of Vascular Inflammation and Atherosclerosis.

79. Because a report in the Journal of the American Medical Association reveals that some Egyptian mummies show signs of atherosclerosis, hardening of the arteries.

80. We investigated the impact of antihypertensive drug to prevent CKD incidence in hypertensive patients with advanced atherosclerosis.

81. It can decrease the concentration of HDL-C thus plays an auxo-action for regression of atherosclerosis.

82. Here is occlusive coronary atherosclerosis. The coronary at the left is narrowed by 60 to 70 %.

83. The significant increase in plasm HCY and copper may contributed to atherosclerosis disease and thrombotic disease of grafts.

84. Oranges rich in vitamin C, vitamin P, can increase the body immunity and increases the flexibility of capillaries, reducing blood cholesterol, can prevent hypertension, atherosclerosis.

85. Objective To investigate the relation between carotid atherosclerosis and lacunar infarction in hypertensive patients.

86. Atherosclerosis is a chronic inflammatory disease in which immune mechanisms play a pivotal role.

87. LDL cholesteryl oleate as a predictor for atherosclerosis: evidence from human and animal studies on dietary fat.

88. Increases in work time are positively related to progression of carotid atherosclerosis in middle - aged men.

89. Three aortas are to demonstrate mild, moderate, and severe atherosclerosis from bottom to top.

90. Increases in high-density lipoprotein cholesterol (HDL-C) levels during statin therapy are associated with regression of coronary atherosclerosis, a review of four clinical trials has confirmed.

91. Atherosclerosis ( AS ) is presently one of the most harmful human diseases with highest morbidity.

92. Moreover, it contributes to the elucidation of the pathophysiological mechanisms of atherosclerosis and some metabolic diseases.

93. Conclusions Aortic arch atherosclerosis is an important potential source of cerebral embolic stroke.

94. The causes were penetrating trauma in 22, blunt trauma in 2, iatrogenic injury in 2, atherosclerosis in 6, medical necrosis in 6, and inflammatory in 2 cases.

95. Studies have shown that the higher the level of LDL, the greater the risk of atherosclerosis. Another type of cholesterol package is called HDL (for high-density lipoprotein).

96. Conclusion The imbalance of plasma redox status deviating to oxidation might be implicated in oxidized injury of lipid, intima thickening and atherosclerosis progress.

97. The main causes are atherosclerosis , infection and trauma , incurring by carotid endarterectomy, and the like.

98. Pain develops because there is a narrowing or blockage in the main artery taking blood to the leg due to hardening of the arteries (atherosclerosis).

99. Effect of an organic germanium compound on blood-fat and experimental atherosclerosis model in rabbits are described.

100. Kidney disorders, fibromyalgia and atherosclerosis (the narrowing of arteries due to cholesterol buildup) are all associated with anorgasmia,[http:///atherosclerosis.html] the review found.

101. OBJECTIVE To observe the effect of Zhengan Xifeng decoction on estradiol(E2) and endothelin(ET-1)level in experimental post-menopause atherosclerosis rats.

102. Objective To evaluate the clinical significance of carotid ultrasonography by studying the relationship of the common carotid artery atherosclerosis with coronary heart disease.

103. From a pathophysiological point of view, the Apo B/A1 ratio is more reflective of endothelial dysfunction and atherosclerosis development than the level of low-density lipoprotein (LDL) cholesterol.

104. A striking exception is probucol, which retards atherosclerosis in carotid arteries and restenosis of coronary arteries after angioplasty.

105. The pale yellow lipid streaks aorta are the earliest lesion of atherosclerosis.

106. Objective: To observe the effect of fluvastatin on carotid atherosclerosis in cerebral infarction patients.

107. Objective: To explore the effects of Tongxinluo capsule(TXL) on the atherosclerosis obliterans(ASO) in iliofemoral artery of rabbits.

108. Chronic apoptosis of vascular smooth muscle cells accelerates atherosclerosis and promotes calcification and medial degeneration.

109. Vertebro- basilar artery insufficiency is one of the most common cerebrovascular disease that always occurs in hypertension, atherosclerosis and cervical spondylosis.

110. In recent years chronic infections have been associated with a disease that causes "furring" of the arteries, called atherosclerosis, which is the main cause of heart attacks.

111. Objective: To study the Effect of probucol on vessel endothelial active factors and inflammatory factors of the low-extremity atherosclerosis disease (LEASD) in the aged people.

112. PurposeTo investigate the anti-oxidation action of the effective parts of Zingiber officinale(EPZ) and to analyze the influence on atherosclerosis.

113. The results of this study suggest that probucol, but not fish oil, may inhibit the initiation of lesion formation in the rat model of atherosclerosis.

114. Coronary disease also calls the anemic heart disease, is refers to the coronal atherosclerosis to cause the lumen to cause heart disease which narrowly the cardiac muscle oxygen deficit causes.

115. The results showed thathyperlipoidemia induced not only atherosclerosis but also pneumonia, fatty and hydropic degenerations of liver cells, and response ofmonounclear macrophage system.

116. Mass research results demonstrate that Genetic CETP deficiency is relative to atherosclerosis.

117. Aim To study the effect of paeonol on proliferation of vascular smooth muscle cell in atherosclerosis rabbits.

118. Endothelial dysfunction is critical pathogenic factor in the development of cardiovascular diseases such as atherosclerosis, hypercholesteremia and disseminated intravascular coagulation.

119. Except these, infection with HCMV is closely related with occurrence of physical and mental defects of new-born, atherosclerosis, colonic diverticulitis.

120. Conclusions Anaurosis fugax was interrelated with the degrees of carotid artery stenosis. The blood velocity was decreased due to the carotid atherosclerosis in the ipsilateral eye.

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