(1) Indicated below are the absolute numbers of thymocytes.

(2) The results of associating various combinations of thymocytes and stromal cells are summarized in Table 1.

(3) Thymocyte DNA from eight littermates was cut with Hin dIII and hybridized with probe B-B.

(4) Methods: The activities of mononuclear cells, thymocyte, splenocyte and macrophage were measured by MTT methods.

(5) Title:Short-term induction therapy with anti- thymocyte globulin and delayed use of calcineurin inhibitors in orthotopic liver transplantation.

(6) CONCLUSION: DEX induces mouse thymocyte apoptosis at least partly through ERK pathway, and ERK inhibition has an important biological significance during this process.

(7) CONCLUSION: The process of thymocyte apoptosis mediated by NO in mice is successively PS ectropion, mitochondrial depolarization, CL oxidation and cell crenation.

(8) Conclusion Cyclophosphamide has impact on thymocyte subpopulation, hydrocortisone has impact on peripheral blood and spleen T lymphocytes.

(9) Conclusion The apoptosis in thymocyte can be induced by Klebsiella pneumoniae infection and the treatment of cefodizime and amikacin can significantly inhibit the thymocyte apoptosis.

(10) Objective To prepare rabbit anti -rat thymocyte serum(ATS) through immunizing New Zealand Rabbit with rats thymocyte cell suspension.

(11) Results: PF18-3 depressed the enhancement of thymocyte apoptosis by MTSC4.

(12) Objective To study the morphologic changes of rat thymocyte apoptosis induced by diabetes and the possible mechanism of thymic involution in diabetes.

(13) Methods:immunity indexes monitoring, experiment of the transformation of the lymphocyte and the auto proliferation reaction of the thymocyte.

(14) Objective To study on the dexamethasone(DEX)-induced mitochondrial depolarization and apoptosis process in mouse thymocyte.

(15) The bulk of the data presented herein suggests that thymocyte may promote MTEC1 proliferation by cell cell interaction and producing cytokine.

(16) Objective To investigate the relationship between PF18 3 McAb recognized molecule and thymocyte activation induced apoptosis.

(17) Objective To study the morphologic changes with age in thymocytes and to explore the effect of luteolin on the aging changes in thymocyte morphology, immunity and anti-oxidation activity in aged mice.

(18) Objective: To study the expression and physiological action of death signal pathway in thymocyte apoptosis in rats.

(19) Objective To explore the morphologic changes of the epithelial cells in the thymic cortex in diabetes rats, and its possible mechanism of thymocyte apoptosis and thymus involution.

(20) Objective To observe the dose-rate effects of adaptive response of thymocyte DNA lysis induced by low dose radiation.

(21) Objective:To observe the dose rate effect of adaptive response of thymocyte apoptosis and cell cycle progression induced by low dose X ray radiation.

(22) AIM: To study the effect of ERK inhibition on the mitochondrial potential change in dexamethasone (DEX)-induced thymocyte apoptosis.

(23) Conclusion: Cyclophosphamide may causes immunosuppression through the induction of apoptosis of thymocyte.

(24) Objective To investigate the effect of the dexamethasone on apoptosis of thymocyte in experimental allergic encephalomyelitis(EAE) mice.