(1) CONCLUSION:Endotoxic hepatic injury is related with the progressively decreased expression of SR-A in liver tissue during ACST.

(2) Methods: tPNS was injected to rats with endotoxic shock and their blood pressure, heart rates, left ventricular stroke volume were observed.

(3) Objective: To investigate the influence of Total Panax Notoginseng Saponins (tPNS) on hemodynamics of rats with endotoxic shock and its mechanism.

(4) Objective To explore the changes of vascular reactivity following endotoxic shock and its relation with hemodynamic changes.

(5) The results suggest that GPs have a significant protective effect against endotoxic shock and prevent from subsequent DIC.

(6) Conclusion NO may have beneficial effect on the outcome of endotoxic shock in the study and inhibition of NO production may be detrimental.

(7) Methods The concentration of serum of ET and TNF of 20 critically ill patients, 18 non-critically ill patients, 10 rats with endotoxic shock and 10 normal rats were tested.

(8) METH0DS: Apply the coacervation mechanism of limulus reagent and bacteria endotoxin to judge it endotoxic limes in specimen was corresponding to the prescript.

(9) The effects of chloroquine and dexamethasone, inhibitors to phospholipase A2(PLA2), on the activity of blood PLA2 and its related lipid mediators were observed in rabbits with endotoxic shock.

(10) Objective: To investigate the effects of MCI 154, a calcium sensitizer, on myocardial calcium homeostasis during endotoxic shock in rats.

(11) Objective: To investigate the ultrastructural changes of thoracic aortic smooth muscle cells in rats with endotoxic shock and to explore the mechanism of vascular hyporeactivity.

(12) Effect of polydatin on phospholipase A2 in lung tissues in rats with endotoxic shock.

(13) Rats were challenged by E. coli LPS to set up the model of endotoxic shock, AG group were treated by aminoguanidine.

(14) Objective: To observe the effects of MCI 154, a calcium sensitizer, on cardiac function after endotoxic shock.

(15) Intestine is an important organ that maintains the nutrition and the immunity in the body, and it is also the biggest bacterial and endotoxic storeroom.

(16) OBJECTIVE: To study the pathogenesis of kidney injury in early phase of endotoxic shock.

(17) Objective: To investigate the effects of MCI 154, a calcium sensitizer, on organ blood flows, arterial blood gases, some plasma biochemical parameters and survival rate in endotoxic shock rabbits.

(18) Objective:To study the mechanism underlying rhubarb inflammatory mediator response in endotoxic shock.

(19) This article reviews the proteins and transduction pat hw ay in endotoxic action.